Injury-dependent Müller glia and ganglion cell reprogramming during tissue regeneration requires Apobec2a and Apobec2b. [electronic resource]
Producer: 20120309Description: 1096-109 p. digitalISSN:- 1529-2401
- Animals
- Animals, Genetically Modified
- Basic Helix-Loop-Helix Transcription Factors -- genetics
- Bromodeoxyuridine -- metabolism
- Cell Death -- genetics
- Cytidine Deaminase -- classification
- ELAV Proteins -- genetics
- Embryo, Nonmammalian
- Enzyme Inhibitors -- pharmacology
- Female
- Gene Expression Regulation -- genetics
- Green Fluorescent Proteins -- genetics
- In Situ Nick-End Labeling
- In Vitro Techniques
- Male
- Neurogenesis -- drug effects
- Neuroglia -- physiology
- Oligonucleotides -- pharmacology
- Optic Nerve Diseases -- complications
- Proliferating Cell Nuclear Antigen -- metabolism
- RNA, Messenger -- metabolism
- Regeneration -- drug effects
- Retina -- injuries
- Retinal Ganglion Cells -- physiology
- Time Factors
- Transcription Factors
- Zebrafish
- Zebrafish Proteins -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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