Sexually dimorphic testosterone secretion in prenatal and neonatal mice is independent of kisspeptin-Kiss1r and GnRH signaling. [electronic resource]
Producer: 20120327Description: 782-93 p. digitalISSN:- 1945-7170
- Animals
- Animals, Newborn -- metabolism
- Female
- Fetus -- metabolism
- Gene Expression Regulation, Developmental -- physiology
- Gonadotropin-Releasing Hormone -- genetics
- Kisspeptins -- genetics
- Male
- Mice
- Mice, Knockout
- Neurokinin B -- genetics
- Receptors, G-Protein-Coupled -- genetics
- Receptors, Kisspeptin-1
- Sex Characteristics
- Signal Transduction
- Testis -- growth & development
- Testosterone -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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