Depletion of endogenous noradrenaline does not prevent spinal cord plasticity following peripheral nerve injury. [electronic resource]
Producer: 20120424Description: 49-57 p. digitalISSN:- 1528-8447
- Acetylcholine -- metabolism
- Adjuvants, Anesthesia -- pharmacology
- Analgesics -- therapeutic use
- Animals
- Anterior Horn Cells -- drug effects
- Atropine -- pharmacology
- Brain-Derived Neurotrophic Factor -- metabolism
- Calcium-Binding Proteins -- metabolism
- Choline O-Acetyltransferase -- metabolism
- Clonidine -- therapeutic use
- Disease Models, Animal
- Dopamine beta-Hydroxylase -- metabolism
- Functional Laterality
- Gene Expression Regulation -- drug effects
- Glial Fibrillary Acidic Protein -- metabolism
- Immunotoxins -- pharmacology
- Male
- Microfilament Proteins -- metabolism
- Norepinephrine -- deficiency
- Pain Measurement
- Pain Threshold -- drug effects
- Peripheral Nerve Injuries -- drug therapy
- Rats
- Rats, Sprague-Dawley
- Ribosome Inactivating Proteins, Type 1 -- pharmacology
- Saporins
- Spinal Cord -- drug effects
- Spinal Nerves -- physiopathology
- Synaptosomes -- drug effects
- Time Factors
- Tritium -- metabolism
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural
There are no comments on this title.
Log in to your account to post a comment.