Radiation-induced increase in cell migration and metastatic potential of cervical cancer cells operates via the K-Ras pathway. [electronic resource]
Producer: 20120325Description: 862-71 p. digitalISSN:- 1525-2191
- Animals
- Cell Movement -- radiation effects
- Female
- Lung Neoplasms -- secondary
- MAP Kinase Signaling System -- radiation effects
- Mice
- Mice, Inbred BALB C
- Mice, Nude
- Neoplasm Metastasis
- Neoplasm Recurrence, Local -- metabolism
- Neoplasms, Radiation-Induced -- metabolism
- Phosphorylation
- Proto-Oncogene Proteins c-raf -- metabolism
- Radiation Tolerance -- radiation effects
- Tumor Cells, Cultured
- Uterine Cervical Neoplasms -- metabolism
- Wound Healing -- radiation effects
- p38 Mitogen-Activated Protein Kinases -- physiology
- ras Proteins -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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