Amyloid-β oligomers stimulate microglia through a tyrosine kinase dependent mechanism. [electronic resource]
Producer: 20130121Description: 2247-61 p. digitalISSN:- 1558-1497
- Alzheimer Disease -- drug therapy
- Amyloid beta-Peptides -- metabolism
- Animals
- Brain -- drug effects
- Cells, Cultured
- Dasatinib
- Female
- Humans
- Male
- Mice
- Mice, Inbred C57BL
- Microglia -- drug effects
- Phosphotyrosine -- analysis
- Plaque, Amyloid -- metabolism
- Polymers -- metabolism
- Protein Kinase Inhibitors -- metabolism
- Protein-Tyrosine Kinases -- antagonists & inhibitors
- Proto-Oncogene Proteins pp60(c-src) -- metabolism
- Pyrimidines -- pharmacology
- Thiazoles -- pharmacology
- Tumor Necrosis Factor-alpha -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
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