A signaling mechanism coupling netrin-1/deleted in colorectal cancer chemoattraction to SNARE-mediated exocytosis in axonal growth cones. [electronic resource]
Producer: 20111206Description: 14463-80 p. digitalISSN:- 1529-2401
- Analysis of Variance
- Animals
- Animals, Newborn
- Axons -- drug effects
- Boron Compounds -- metabolism
- Botulinum Toxins, Type A -- pharmacology
- Brain-Derived Neurotrophic Factor -- pharmacology
- Cells, Cultured
- Chemotaxis -- drug effects
- Chlorocebus aethiops
- Complement C1 -- pharmacology
- DCC Receptor
- Embryo, Mammalian
- Exocytosis -- drug effects
- Gene Expression Regulation, Developmental -- drug effects
- Green Fluorescent Proteins -- genetics
- Growth Cones -- drug effects
- Guanine Nucleotide Exchange Factors -- metabolism
- Hippocampus -- cytology
- Humans
- Immunoprecipitation
- Mice
- Mice, Knockout
- Mice, Transgenic
- Munc18 Proteins -- genetics
- Nerve Growth Factors -- genetics
- Nerve Tissue Proteins -- metabolism
- Netrin-1
- Neuromuscular Agents -- pharmacology
- Neurons -- cytology
- Organ Culture Techniques
- Receptors, Cell Surface -- genetics
- SNARE Proteins -- genetics
- Signal Transduction -- drug effects
- Surface Plasmon Resonance -- methods
- Tetanus Toxin -- pharmacology
- Transfection -- methods
- Tumor Suppressor Proteins -- genetics
- Vesicle-Associated Membrane Protein 2 -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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