Absence of SOD1 leads to oxidative stress in peripheral nerve and causes a progressive distal motor axonopathy. [electronic resource]
Producer: 20120325Description: 163-71 p. digitalISSN:- 1090-2430
- Acetylcysteine -- pharmacology
- Age Factors
- Analysis of Variance
- Animals
- Axons -- pathology
- Bacterial Proteins -- genetics
- Calcium-Binding Proteins -- metabolism
- Chromatography, High Pressure Liquid
- DNA-Binding Proteins -- metabolism
- Free Radical Scavengers -- pharmacology
- Glial Fibrillary Acidic Protein -- metabolism
- Glutathione -- metabolism
- Glutathione Disulfide -- metabolism
- Luminescent Proteins -- genetics
- Mice
- Mice, Knockout
- Microfilament Proteins -- metabolism
- Muscle Denervation -- methods
- Muscle, Skeletal -- physiopathology
- Nerve Fibers -- pathology
- Neuromuscular Junction -- physiopathology
- Oxidation-Reduction -- drug effects
- Oxidative Stress -- genetics
- Peripheral Nerves -- metabolism
- Peripheral Nervous System Diseases -- pathology
- Presynaptic Terminals -- metabolism
- Skin -- innervation
- Superoxide Dismutase -- deficiency
- Superoxide Dismutase-1
- Ubiquitin Thiolesterase -- metabolism
- Ubiquitins -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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