Inhibition of soluble tumour necrosis factor is therapeutic in experimental autoimmune encephalomyelitis and promotes axon preservation and remyelination. [electronic resource]
Producer: 20111108Description: 2736-54 p. digitalISSN:- 1460-2156
- Animals
- Axons -- immunology
- Chemokines -- immunology
- Cytokines -- immunology
- Encephalomyelitis, Autoimmune, Experimental -- immunology
- Female
- Glycoproteins -- immunology
- Humans
- Leukocytes -- cytology
- Mice
- Mice, Inbred C57BL
- Multiple Sclerosis -- immunology
- Myelin Sheath -- metabolism
- Myelin-Oligodendrocyte Glycoprotein
- Nerve Regeneration -- physiology
- Peptide Fragments -- immunology
- Receptors, Tumor Necrosis Factor -- metabolism
- Spinal Cord -- cytology
- Tumor Necrosis Factor Inhibitors
- Tumor Necrosis Factors -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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