Frequent EVI1 translocations in myeloid blast crisis CML that evolves through tyrosine kinase inhibitors. [electronic resource]
Producer: 20111021Description: 392-7 p. digitalISSN:- 2210-7762
- Adult
- Aged
- Blast Crisis -- genetics
- Blotting, Western
- DNA Breaks, Double-Stranded -- drug effects
- DNA Repair
- DNA-Binding Proteins -- genetics
- Drug Resistance, Neoplasm -- genetics
- Evolution, Molecular
- Female
- Fusion Proteins, bcr-abl -- genetics
- Genes, abl
- Humans
- In Situ Hybridization, Fluorescence
- Leukemia, Myelogenous, Chronic, BCR-ABL Positive -- pathology
- MDS1 and EVI1 Complex Locus Protein
- Male
- Middle Aged
- Mutation
- Protein Kinase Inhibitors -- therapeutic use
- Protein-Tyrosine Kinases -- metabolism
- Proto-Oncogenes -- genetics
- Transcription Factors -- genetics
- Translocation, Genetic
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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