Deletion of UCP2 in iNOS deficient mice reduces the severity of the disease during experimental autoimmune encephalomyelitis. [electronic resource]
Producer: 20120215Description: e22841 p. digitalISSN:- 1932-6203
- Animals
- Brain -- metabolism
- Cytokines -- genetics
- Encephalomyelitis, Autoimmune, Experimental -- genetics
- Glutathione -- metabolism
- Glutathione Disulfide -- metabolism
- Humans
- Immune System -- metabolism
- Ion Channels -- deficiency
- Macrophages, Peritoneal -- metabolism
- Mice
- Mice, Knockout
- Mitochondrial Proteins -- deficiency
- Nitric Oxide -- metabolism
- Nitric Oxide Synthase Type II -- deficiency
- Oxidation-Reduction
- Oxidative Stress
- Reactive Oxygen Species -- metabolism
- Reverse Transcriptase Polymerase Chain Reaction
- Severity of Illness Index
- Spinal Cord -- metabolism
- Uncoupling Protein 2
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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