Shedding of the Mer tyrosine kinase receptor is mediated by ADAM17 protein through a pathway involving reactive oxygen species, protein kinase Cδ, and p38 mitogen-activated protein kinase (MAPK). [electronic resource]
Producer: 20111123Description: 33335-44 p. digitalISSN:- 1083-351X
- ADAM Proteins -- chemistry
- ADAM17 Protein
- Adaptor Proteins, Vesicular Transport -- metabolism
- Amino Acid Sequence
- Animals
- Lipopolysaccharides -- pharmacology
- Mass Spectrometry
- Mice
- Mice, Inbred C57BL
- Molecular Sequence Data
- Myeloid Differentiation Factor 88 -- metabolism
- NADP -- deficiency
- Proline -- metabolism
- Protein Kinase C-delta -- metabolism
- Protein Processing, Post-Translational -- drug effects
- Proto-Oncogene Proteins -- metabolism
- Reactive Oxygen Species -- metabolism
- Receptor Protein-Tyrosine Kinases -- metabolism
- Sequence Deletion
- Signal Transduction -- drug effects
- Solubility -- drug effects
- Toll-Like Receptor 4 -- metabolism
- c-Mer Tyrosine Kinase
- p38 Mitogen-Activated Protein Kinases -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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