The VPAC2 agonist peptide histidine isoleucine (PHI) up-regulates glutamate transport in the corpus callosum of a rat model of amyotrophic lateral sclerosis (hSOD1G93A) by inhibiting caspase-3 mediated inactivation of GLT-1a. [electronic resource]
Producer: 20111207Description: 3674-86 p. digitalISSN:- 1530-6860
- Amyotrophic Lateral Sclerosis -- metabolism
- Animals
- Animals, Genetically Modified
- Biological Transport
- Caspase 3 -- metabolism
- Corpus Callosum -- drug effects
- Excitatory Amino Acid Transporter 2 -- metabolism
- Gene Expression Regulation
- Glial Fibrillary Acidic Protein -- genetics
- Glutamates -- metabolism
- Myelin Proteins -- genetics
- Myelin-Oligodendrocyte Glycoprotein
- Peptide PHI -- pharmacology
- Pituitary Adenylate Cyclase-Activating Polypeptide
- Rats
- Superoxide Dismutase -- genetics
- Superoxide Dismutase-1
- Vasoactive Intestinal Peptide
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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