Vascular β-amyloid and early astrocyte alterations impair cerebrovascular function and cerebral metabolism in transgenic arcAβ mice. [electronic resource]
Producer: 20120110Description: 293-311 p. digitalISSN:- 1432-0533
- Amyloid beta-Peptides -- genetics
- Amyloid beta-Protein Precursor -- metabolism
- Animals
- Astrocytes -- drug effects
- Basement Membrane -- metabolism
- Blood-Brain Barrier -- pathology
- Brain -- pathology
- Cell Culture Techniques
- Cerebral Amyloid Angiopathy -- complications
- Cerebral Arteries -- metabolism
- Cerebrovascular Disorders -- etiology
- Disease Models, Animal
- Disease Progression
- Dystroglycans -- metabolism
- Endothelium -- metabolism
- Gene Expression Regulation -- genetics
- Glial Fibrillary Acidic Protein -- metabolism
- Glucose -- metabolism
- Glucose Transporter Type 1 -- genetics
- Hemorrhage -- etiology
- Humans
- Lactase -- metabolism
- Laminin -- metabolism
- Mice
- Mice, Transgenic
- Microdialysis -- methods
- Microscopy, Electron, Scanning -- methods
- Monocarboxylic Acid Transporters -- genetics
- Muscle, Smooth -- metabolism
- Plaque, Amyloid -- metabolism
- Platelet Endothelial Cell Adhesion Molecule-1 -- metabolism
- Symporters -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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