Attenuating the endoplasmic reticulum stress response improves functional recovery after spinal cord injury. [electronic resource]
Producer: 20111129Description: 1489-502 p. digitalISSN:- 1098-1136
- Activating Transcription Factor 4 -- metabolism
- Analysis of Variance
- Animals
- Apoptosis -- genetics
- Basic Helix-Loop-Helix Transcription Factors -- genetics
- Caspase 3 -- genetics
- DNA-Binding Proteins -- metabolism
- Disease Models, Animal
- Endoplasmic Reticulum Stress -- genetics
- Eukaryotic Initiation Factor-2 -- metabolism
- Female
- Locomotion -- physiology
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Myelin Basic Protein -- genetics
- Nerve Fibers, Myelinated -- metabolism
- Nerve Tissue Proteins -- genetics
- Oligodendrocyte Transcription Factor 2
- Oligodendroglia -- metabolism
- RNA, Messenger -- metabolism
- Recovery of Function -- physiology
- Regulatory Factor X Transcription Factors
- Spinal Cord Injuries -- metabolism
- Time Factors
- Transcription Factor CHOP -- deficiency
- Transcription Factors -- metabolism
- Unfolded Protein Response -- genetics
- Up-Regulation -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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