Loss of the receptor tyrosine kinase Axl leads to enhanced inflammation in the CNS and delayed removal of myelin debris during experimental autoimmune encephalomyelitis. [electronic resource]
Producer: 20111003Description: 49 p. digitalISSN:- 1742-2094
- Animals
- Central Nervous System -- immunology
- Cytokines -- immunology
- Encephalomyelitis, Autoimmune, Experimental -- immunology
- Glycoproteins -- immunology
- Inflammation -- immunology
- Macrophages -- cytology
- Mice
- Mice, Knockout
- Microglia -- cytology
- Myelin Sheath -- metabolism
- Myelin-Oligodendrocyte Glycoprotein
- Peptide Fragments -- immunology
- Proto-Oncogene Proteins -- genetics
- Receptor Protein-Tyrosine Kinases -- genetics
- Axl Receptor Tyrosine Kinase
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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