Aging-induced collateral dysfunction: impaired responsiveness of collaterals and susceptibility to apoptosis via dysfunctional eNOS signaling. [electronic resource]
Producer: 20120312Description: 779-89 p. digitalISSN:- 1937-5395
- Age Factors
- Aging -- metabolism
- Animals
- Aorta -- enzymology
- Apoptosis
- Arterial Occlusive Diseases -- enzymology
- Blotting, Western
- Bone Marrow Transplantation
- Cell Movement
- Cells, Cultured
- Collateral Circulation
- Cyclic Nucleotide Phosphodiesterases, Type 5 -- metabolism
- Disease Models, Animal
- Endothelial Cells -- enzymology
- Female
- Femoral Artery -- surgery
- Laser-Doppler Flowmetry
- Ligation
- Lower Extremity -- blood supply
- Mice
- Mice, Inbred C57BL
- Muscle, Smooth, Vascular -- enzymology
- Myocytes, Smooth Muscle -- enzymology
- Neovascularization, Physiologic
- Nitric Oxide Synthase Type III -- metabolism
- Perfusion Imaging -- methods
- Phosphorylation
- Regional Blood Flow
- Signal Transduction
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural
There are no comments on this title.
Log in to your account to post a comment.