The response to TLR ligation of human CD16⁺CD14⁻ monocytes is weakly modulated as a consequence of persistent infection with the hepatitis C virus. [electronic resource]
Producer: 20110810Description: 1505-11 p. digitalISSN:- 1872-9142
- Adult
- Cell Count
- Chemokine CCL4 -- biosynthesis
- Cytokines -- biosynthesis
- Female
- Hepacivirus -- immunology
- Hepatitis C, Chronic -- immunology
- Humans
- Imidazoles -- pharmacology
- Lipopolysaccharide Receptors -- immunology
- Lipopolysaccharides -- immunology
- Male
- Middle Aged
- Monocytes -- immunology
- Receptors, IgG -- immunology
- Toll-Like Receptor 4 -- metabolism
- Toll-Like Receptor 8 -- agonists
- Tumor Necrosis Factor-alpha -- biosynthesis
No physical items for this record
Publication Type: Journal Article
There are no comments on this title.
Log in to your account to post a comment.