Switching of N-methyl-D-aspartate (NMDA) receptor-favorite intracellular signal pathways from ERK1/2 protein to p38 mitogen-activated protein kinase leads to developmental changes in NMDA neurotoxicity. [electronic resource]
Producer: 20110805Description: 20175-93 p. digitalISSN:- 1083-351X
- Aging -- physiology
- Animals
- Calcium -- metabolism
- Cell Differentiation -- physiology
- Embryo, Mammalian -- metabolism
- Enzyme Activation -- physiology
- Hippocampus -- cytology
- MAP Kinase Signaling System -- physiology
- Male
- Mitogen-Activated Protein Kinase 3 -- genetics
- N-Methylaspartate -- genetics
- Neurons -- cytology
- Rats
- Rats, Sprague-Dawley
- Receptors, N-Methyl-D-Aspartate -- genetics
- p38 Mitogen-Activated Protein Kinases -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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