Impairment of the programmed cell death-1 pathway increases atherosclerotic lesion development and inflammation. [electronic resource]
Producer: 20110624Description: 1100-7 p. digitalISSN:- 1524-4636
- Animals
- Antibodies, Blocking -- pharmacology
- Antigens, Surface -- genetics
- Apoptosis
- Apoptosis Regulatory Proteins -- antagonists & inhibitors
- Atherosclerosis -- genetics
- B7-1 Antigen -- genetics
- B7-H1 Antigen
- Bone Marrow Transplantation
- CD3 Complex -- metabolism
- CD4-Positive T-Lymphocytes -- immunology
- CD8-Positive T-Lymphocytes -- immunology
- Cell Proliferation
- Cells, Cultured
- Disease Models, Animal
- Genes, T-Cell Receptor
- Genotype
- Hypercholesterolemia -- genetics
- Inflammation -- genetics
- Lipoproteins, LDL -- metabolism
- Lymph Nodes -- immunology
- Lymphocyte Activation -- drug effects
- Macrophages -- immunology
- Male
- Membrane Glycoproteins -- deficiency
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Peptides -- deficiency
- Phenotype
- Programmed Cell Death 1 Ligand 2 Protein
- Programmed Cell Death 1 Receptor
- Receptors, LDL -- deficiency
- Signal Transduction
- T-Lymphocyte Subsets -- drug effects
- T-Lymphocytes, Cytotoxic -- immunology
- Time Factors
- Tumor Necrosis Factor-alpha -- blood
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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