Early inflammatory reactions in atherosclerosis are induced by proline-rich tyrosine kinase/reactive oxygen species-mediated release of tumor necrosis factor-alpha and subsequent activation of the p21Cip1/Ets-1/p300 system. [electronic resource]
Producer: 20110624Description: 1084-92 p. digitalISSN:- 1524-4636
- Animals
- Aortic Diseases -- enzymology
- Apolipoproteins E -- deficiency
- Atherosclerosis -- enzymology
- Bone Marrow Transplantation
- Cells, Cultured
- Chemokine CCL2 -- metabolism
- Cyclin-Dependent Kinase Inhibitor p21 -- genetics
- Disease Models, Animal
- Endothelial Cells -- enzymology
- Focal Adhesion Kinase 2 -- deficiency
- Hypercholesterolemia -- enzymology
- Inflammation Mediators -- metabolism
- Lipoproteins, LDL -- metabolism
- Lysophosphatidylcholines -- metabolism
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- NADPH Oxidases -- metabolism
- Proto-Oncogene Protein c-ets-1 -- genetics
- RNA Interference
- Reactive Oxygen Species -- metabolism
- Signal Transduction
- Time Factors
- Transcriptional Activation
- Transfection
- Tumor Necrosis Factor-alpha -- metabolism
- Vascular Cell Adhesion Molecule-1 -- metabolism
- p300-CBP Transcription Factors -- genetics
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't; Retracted Publication
There are no comments on this title.
Log in to your account to post a comment.