Role of extracellular signal-regulated kinase in synaptic transmission and plasticity of a nociceptive input on capsular central amygdaloid neurons in normal and acid-induced muscle pain mice. [electronic resource]
Producer: 20110325Description: 2258-70 p. digitalISSN:- 1529-2401
- Acids -- adverse effects
- Afferent Pathways -- physiology
- Amygdala -- pathology
- Animals
- Disease Models, Animal
- Dose-Response Relationship, Drug
- Drug Interactions
- Electric Stimulation
- Enzyme Inhibitors -- pharmacology
- Excitatory Amino Acid Agents -- pharmacology
- Excitatory Postsynaptic Potentials -- drug effects
- Extracellular Signal-Regulated MAP Kinases -- metabolism
- Female
- Gene Expression Regulation, Enzymologic -- drug effects
- In Vitro Techniques
- Long-Term Potentiation -- drug effects
- Male
- Mice
- Mice, Inbred C57BL
- Muscle, Skeletal -- physiopathology
- Pain -- chemically induced
- Pain Measurement
- Patch-Clamp Techniques -- methods
- Phorbol Esters -- pharmacology
- Phosphorylation
- Sensory Receptor Cells -- cytology
- Sodium Channel Blockers -- pharmacology
- Synaptic Transmission -- physiology
- Tetrodotoxin -- pharmacology
- Time Factors
- Ventral Tegmental Area -- drug effects
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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