Strict 3' splice site sequence requirements for U2 snRNP recruitment after U2AF binding underlie a genetic defect leading to autoimmune disease. [electronic resource]
Producer: 20110404Description: 401-11 p. digitalISSN:- 1469-9001
- Autoimmune Lymphoproliferative Syndrome -- etiology
- Cell Nucleus -- genetics
- Cross-Linking Reagents -- pharmacology
- HeLa Cells
- Humans
- Immunoprecipitation
- Mutation -- genetics
- Nuclear Proteins -- genetics
- RNA Precursors -- genetics
- RNA Splicing -- genetics
- Ribonucleoprotein, U2 Small Nuclear -- genetics
- Ribonucleoproteins -- genetics
- Splicing Factor U2AF
- fas Receptor -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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