Increased susceptibility to acute kidney injury due to endoplasmic reticulum stress in mice lacking tumor necrosis factor-α and its receptor 1. [electronic resource]
Producer: 20110613Description: 613-623 p. digitalISSN:- 1523-1755
- Acute Kidney Injury -- chemically induced
- Animals
- Apoptosis
- Cells, Cultured
- Disease Models, Animal
- Disease Susceptibility
- Endoplasmic Reticulum -- drug effects
- Enzyme Inhibitors -- pharmacology
- Eukaryotic Initiation Factor-2 -- metabolism
- Female
- Kidney Tubules, Proximal -- drug effects
- Lipid Metabolism
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Phosphoprotein Phosphatases -- antagonists & inhibitors
- Phosphorylation
- Receptors, Tumor Necrosis Factor, Type I -- deficiency
- Receptors, Tumor Necrosis Factor, Type II -- deficiency
- Severity of Illness Index
- Signal Transduction
- Time Factors
- Tumor Necrosis Factor-alpha -- deficiency
- Tunicamycin
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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