Elevated AKR1C3 expression promotes prostate cancer cell survival and prostate cell-mediated endothelial cell tube formation: implications for prostate cancer progression. [electronic resource]
Producer: 20110331Description: 672 p. digitalISSN:- 1471-2407
- 3-Hydroxysteroid Dehydrogenases -- genetics
- Blotting, Western
- Cell Line, Tumor
- Cell Survival
- Computational Biology
- Dihydrotestosterone -- metabolism
- Disease Progression
- Endothelial Cells -- drug effects
- Enzyme-Linked Immunosorbent Assay
- Estradiol -- metabolism
- Gene Expression Profiling -- methods
- Gene Expression Regulation, Neoplastic
- Gene Regulatory Networks
- Humans
- Hydroxyprostaglandin Dehydrogenases -- genetics
- Insulin-Like Growth Factor I -- metabolism
- Male
- Neovascularization, Pathologic -- enzymology
- Oligonucleotide Array Sequence Analysis
- Promoter Regions, Genetic
- Prostatic Neoplasms -- blood supply
- Protein Kinase Inhibitors -- pharmacology
- Proto-Oncogene Proteins c-akt -- antagonists & inhibitors
- Receptor, IGF Type 1 -- antagonists & inhibitors
- Receptors, Calcitriol -- metabolism
- Retinoid X Receptors -- metabolism
- Reverse Transcriptase Polymerase Chain Reaction
- Signal Transduction
- Transfection
- Tumor Suppressor Protein p53 -- metabolism
- Up-Regulation
- Vascular Endothelial Growth Factor A -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
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