Neuroprotection by histone deacetylase-7 (HDAC7) occurs by inhibition of c-jun expression through a deacetylase-independent mechanism. [electronic resource]
Producer: 20110324Description: 4819-28 p. digitalISSN:- 1083-351X
- Animals
- Apoptosis -- drug effects
- Cell Line, Tumor
- Cerebellum -- cytology
- Extracellular Signal-Regulated MAP Kinases -- antagonists & inhibitors
- Female
- Gene Expression Regulation -- drug effects
- Histone Deacetylase Inhibitors -- pharmacology
- Histone Deacetylases -- genetics
- MAP Kinase Kinase Kinases -- antagonists & inhibitors
- MAP Kinase Signaling System -- drug effects
- Male
- Mice
- Mice, Transgenic
- Neurons -- cytology
- Phosphatidylinositol 3-Kinases -- metabolism
- Phosphoinositide-3 Kinase Inhibitors
- Protein Kinase Inhibitors -- pharmacology
- Protein Structure, Tertiary
- Proto-Oncogene Proteins c-akt -- antagonists & inhibitors
- Proto-Oncogene Proteins c-jun -- biosynthesis
- Rats
- Rats, Wistar
- Transcription, Genetic -- drug effects
No physical items for this record
Publication Type: Journal Article; Research Support, N.I.H., Extramural
There are no comments on this title.
Log in to your account to post a comment.