Hepcidin as a therapeutic tool to limit iron overload and improve anemia in β-thalassemic mice. [electronic resource]
Producer: 20110114Description: 4466-77 p. digitalISSN:- 1558-8238
- Animals
- Antimicrobial Cationic Peptides -- genetics
- Base Sequence
- DNA Primers -- genetics
- Disease Models, Animal
- Erythropoiesis -- drug effects
- Gene Expression
- Hepcidins
- Humans
- Iron -- metabolism
- Iron Overload -- blood
- Iron, Dietary -- administration & dosage
- Mice
- Mice, Inbred C57BL
- Mice, Mutant Strains
- Mice, Transgenic
- Recombinant Proteins -- genetics
- beta-Thalassemia -- blood
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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