Deletion of EP4 on bone marrow-derived cells enhances inflammation and angiotensin II-induced abdominal aortic aneurysm formation. [electronic resource]
Producer: 20110314Description: 261-9 p. digitalISSN:- 1524-4636
- Angiotensin II -- adverse effects
- Animals
- Aortic Aneurysm, Abdominal -- chemically induced
- Bone Marrow Cells -- cytology
- Bone Marrow Transplantation
- Chemokine CCL2 -- metabolism
- Elastin -- metabolism
- Female
- Gene Deletion
- Hyperlipidemias -- complications
- Inflammation -- chemically induced
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Models, Animal
- Receptors, LDL -- genetics
- Receptors, Prostaglandin E, EP4 Subtype -- genetics
- Risk Factors
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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