An increase in persistent sodium current contributes to intrinsic neuronal bursting after status epilepticus. [electronic resource]
Producer: 20110617Description: 117-29 p. digitalISSN:- 1522-1598
- Animals
- CA1 Region, Hippocampal -- cytology
- Calcium Channel Blockers -- pharmacology
- Calcium Channels, T-Type -- drug effects
- Male
- Models, Animal
- Neurons -- drug effects
- Patch-Clamp Techniques
- Pilocarpine -- adverse effects
- Rats
- Rats, Inbred Strains
- Rats, Wistar
- Sodium Channel Blockers -- pharmacology
- Sodium Channels -- drug effects
- Status Epilepticus -- chemically induced
- Tetrodotoxin -- pharmacology
- Time Factors
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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