Developmental defects and neuromuscular alterations due to mitofusin 2 gene (MFN2) silencing in zebrafish: a new model for Charcot-Marie-Tooth type 2A neuropathy. [electronic resource]
Producer: 20110421Description: 58-67 p. digitalISSN:- 1873-2364
- Animals
- Animals, Genetically Modified
- Body Patterning -- drug effects
- Charcot-Marie-Tooth Disease -- chemically induced
- Computational Biology
- Developmental Disabilities -- etiology
- Disease Models, Animal
- Down-Regulation -- drug effects
- Embryo, Nonmammalian
- GTP Phosphohydrolases
- Humans
- Membrane Proteins -- genetics
- Mitochondrial Proteins -- genetics
- Motor Activity -- drug effects
- Neuromuscular Diseases -- etiology
- Oligodeoxyribonucleotides, Antisense -- adverse effects
- RNA, Messenger -- metabolism
- Receptors, Cholinergic -- metabolism
- Tolloid-Like Metalloproteinases -- genetics
- Tubulin -- metabolism
- Zebrafish
- Zebrafish Proteins -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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