Mutations in cytoplasmic dynein lead to a Huntington's disease-like defect in energy metabolism of brown and white adipose tissues. [electronic resource]
Producer: 20110307Description: 59-69 p. digitalISSN:- 0006-3002
- 3T3-L1 Cells
- Adipose Tissue, Brown -- metabolism
- Adipose Tissue, White -- metabolism
- Adrenergic alpha-Agonists -- pharmacology
- Animals
- Blotting, Western
- Cytoplasmic Dyneins -- genetics
- Energy Metabolism -- genetics
- Female
- Gene Expression
- Humans
- Huntingtin Protein
- Huntington Disease -- genetics
- Lipolysis -- drug effects
- Male
- Mice
- Mice, Mutant Strains
- Mutation
- Nerve Tissue Proteins -- genetics
- Norepinephrine -- pharmacology
- Nuclear Proteins -- genetics
- Oxidative Stress -- drug effects
- Receptors, Adrenergic, beta-2 -- metabolism
- Reverse Transcriptase Polymerase Chain Reaction
- Signal Transduction
- Thermogenesis -- genetics
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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