Epidermal growth factor receptor transactivation by endogenous vasoactive peptides contributes to hyperproliferation of vascular smooth muscle cells of SHR. [electronic resource]
Producer: 20110113Description: H1959-67 p. digitalISSN:- 1522-1539
- Angiotensin II -- metabolism
- Angiotensin II Type 1 Receptor Blockers -- pharmacology
- Animals
- CSK Tyrosine-Protein Kinase
- Cell Proliferation -- drug effects
- Cells, Cultured
- Disease Models, Animal
- Endothelin A Receptor Antagonists
- Endothelin B Receptor Antagonists
- Endothelin-1 -- metabolism
- Enzyme Inhibitors -- pharmacology
- Epidermal Growth Factor -- antagonists & inhibitors
- ErbB Receptors -- metabolism
- Free Radical Scavengers -- pharmacology
- Hyperplasia
- Hypertension -- metabolism
- Mitogen-Activated Protein Kinases -- metabolism
- Muscle, Smooth, Vascular -- drug effects
- Myocytes, Smooth Muscle -- drug effects
- NADPH Oxidases -- antagonists & inhibitors
- Oxidative Stress
- Phosphorylation
- Protein-Tyrosine Kinases -- antagonists & inhibitors
- Rats
- Rats, Inbred SHR
- Rats, Inbred WKY
- Receptor, Angiotensin, Type 1 -- drug effects
- Receptor, Endothelin A -- metabolism
- Receptor, Endothelin B -- metabolism
- Signal Transduction
- Superoxides -- metabolism
- src-Family Kinases
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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