Loss of PI3Kγ enhances cAMP-dependent MMP remodeling of the myocardial N-cadherin adhesion complexes and extracellular matrix in response to early biomechanical stress. [electronic resource]
Producer: 20101215Description: 1275-89 p. digitalISSN:- 1524-4571
- Adrenergic beta-Antagonists -- administration & dosage
- Animals
- Antigens, CD -- metabolism
- Biomechanical Phenomena
- Cadherins -- metabolism
- Cardiomegaly -- drug therapy
- Cell Adhesion
- Cells, Cultured
- Class Ib Phosphatidylinositol 3-Kinase -- deficiency
- Cyclic AMP -- metabolism
- Cyclic AMP Response Element-Binding Protein -- metabolism
- Disease Models, Animal
- Extracellular Matrix -- metabolism
- Fibroblasts -- enzymology
- Glycogen Synthase Kinase 3 -- metabolism
- Heart Failure -- enzymology
- Male
- Matrix Metalloproteinase 13 -- metabolism
- Matrix Metalloproteinase 14 -- metabolism
- Matrix Metalloproteinase 2 -- metabolism
- Matrix Metalloproteinases -- metabolism
- Mechanotransduction, Cellular -- drug effects
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- Myocardial Contraction
- Myocardium -- enzymology
- Myocytes, Cardiac -- enzymology
- Phosphorylation
- Propranolol -- administration & dosage
- Proto-Oncogene Proteins c-akt -- metabolism
- Stress, Mechanical
- Time Factors
- Ventricular Remodeling -- drug effects
- beta Catenin -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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