Sevoflurane postconditioning reduces myocardial reperfusion injury in rat isolated hearts via activation of PI3K/Akt signaling and modulation of Bcl-2 family proteins. [electronic resource]
Producer: 20110104Description: 661-72 p. digitalISSN:- 1862-1783
- Androstadienes -- pharmacology
- Anesthetics, Inhalation -- antagonists & inhibitors
- Animals
- Apoptosis -- drug effects
- Blood Pressure -- physiology
- Blotting, Western
- Creatine Kinase -- analysis
- Heart Rate -- physiology
- Immunosuppressive Agents -- pharmacology
- In Vitro Techniques
- Ischemic Postconditioning -- methods
- L-Lactate Dehydrogenase -- analysis
- Male
- Methyl Ethers -- antagonists & inhibitors
- Myocardial Reperfusion Injury -- drug therapy
- Proto-Oncogene Proteins c-akt -- metabolism
- Proto-Oncogene Proteins c-bcl-2 -- metabolism
- Random Allocation
- Rats
- Rats, Sprague-Dawley
- Sevoflurane
- Signal Transduction -- physiology
- Ventricular Pressure -- physiology
- Wortmannin
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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