Endogenous prostaglandin E2 accelerates healing of indomethacin-induced small intestinal lesions through upregulation of vascular endothelial growth factor expression by activation of EP4 receptors. [electronic resource]
Producer: 20101027Description: S67-74 p. digitalISSN:- 1440-1746
- 16,16-Dimethylprostaglandin E2 -- pharmacology
- Animals
- Cyclooxygenase 1 -- genetics
- Cyclooxygenase 2 -- genetics
- Dinoprostone -- metabolism
- Disease Models, Animal
- Gene Expression Regulation, Enzymologic
- Indomethacin
- Intestinal Mucosa -- drug effects
- Intestine, Small -- drug effects
- Male
- Membrane Proteins -- genetics
- Naphthalenes -- pharmacology
- Neovascularization, Physiologic -- drug effects
- Peptic Ulcer -- chemically induced
- Phenylbutyrates -- pharmacology
- RNA, Messenger -- metabolism
- Rats
- Rats, Sprague-Dawley
- Receptors, Prostaglandin E -- agonists
- Receptors, Prostaglandin E, EP4 Subtype
- Severity of Illness Index
- Time Factors
- Up-Regulation
- Vascular Endothelial Growth Factor A -- metabolism
- Wound Healing -- drug effects
No physical items for this record
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
There are no comments on this title.
Log in to your account to post a comment.