Ionic dysregulations typical of ischemia provoke release of glycine and GABA by multiple mechanisms. [electronic resource]
Producer: 20101006Description: 1074-84 p. digitalISSN:- 1471-4159
- Animals
- Brain Ischemia -- metabolism
- Exocytosis -- drug effects
- Glycine -- metabolism
- Hippocampus -- drug effects
- Mice
- Mitochondria -- metabolism
- Neurotransmitter Transport Proteins -- metabolism
- Potassium Chloride -- pharmacology
- Presynaptic Terminals -- drug effects
- Sodium-Calcium Exchanger -- drug effects
- Spinal Cord -- drug effects
- Subcellular Fractions -- drug effects
- Synaptosomes -- drug effects
- Veratridine -- pharmacology
- gamma-Aminobutyric Acid -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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