B7-H1-deficiency enhances the potential of tolerogenic dendritic cells by activating CD1d-restricted type II NKT cells. [electronic resource]
Producer: 20100907Description: e10800 p. digitalISSN:- 1932-6203
- Animals
- Antigens, CD1d -- immunology
- Antigens, Surface -- metabolism
- Apoptosis Regulatory Proteins -- metabolism
- B7-1 Antigen -- metabolism
- B7-H1 Antigen
- Biomarkers -- metabolism
- CD4-Positive T-Lymphocytes -- drug effects
- Cell Line
- Cell Membrane -- drug effects
- Central Nervous System -- drug effects
- Dendritic Cells -- drug effects
- Encephalomyelitis, Autoimmune, Experimental -- immunology
- Epitopes -- immunology
- Lymphocyte Activation -- drug effects
- Membrane Glycoproteins -- deficiency
- Mice
- Natural Killer T-Cells -- drug effects
- Organic Chemicals -- immunology
- Peptides -- deficiency
- Programmed Cell Death 1 Receptor
- Tumor Necrosis Factor-alpha -- administration & dosage
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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