Sexual dimorphic evolution of metabolic programming in non-genetic non-alimentary mild metabolic syndrome model in mice depends on feed-back mechanisms integrity for pro-opiomelanocortin-derived endogenous substances. [electronic resource]
Producer: 20101123Description: 1598-605 p. digitalISSN:- 1873-5169
- 11-beta-Hydroxysteroid Dehydrogenase Type 1 -- genetics
- 11-beta-Hydroxysteroid Dehydrogenase Type 2 -- genetics
- Adrenocorticotropic Hormone -- blood
- Animals
- Animals, Newborn
- Brain -- metabolism
- Corticosterone -- blood
- Diabetes Mellitus, Type 2 -- blood
- Feedback, Physiological
- Female
- Gene Expression Regulation, Enzymologic
- Handling, Psychological
- Hypothalamo-Hypophyseal System -- metabolism
- Lipid Metabolism -- physiology
- Male
- Metabolic Syndrome -- blood
- Mice
- Pain Threshold -- physiology
- Pituitary-Adrenal System -- metabolism
- Pro-Opiomelanocortin -- metabolism
- RNA, Messenger -- metabolism
- Random Allocation
- Sex Characteristics
- Stress, Psychological -- complications
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Publication Type: Journal Article
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