HIV-1 exploits innate signaling by TLR8 and DC-SIGN for productive infection of dendritic cells. [electronic resource]
Producer: 20100817Description: 419-26 p. digitalISSN:- 1529-2916
- Cell Adhesion Molecules -- immunology
- Cells, Cultured
- Dendritic Cells -- immunology
- HIV Envelope Protein gp120 -- genetics
- HIV Infections -- genetics
- HIV-1 -- pathogenicity
- Humans
- Immunity, Innate
- Lectins, C-Type -- immunology
- NF-kappa B -- genetics
- Phosphorylation
- Positive Transcriptional Elongation Factor B -- metabolism
- Protein Binding -- genetics
- Protein Engineering
- Proto-Oncogene Proteins c-raf -- metabolism
- RNA Polymerase II -- metabolism
- Receptors, Cell Surface -- immunology
- Second Messenger Systems -- genetics
- Sequence Deletion -- genetics
- Toll-Like Receptor 8 -- immunology
- Transcriptional Activation -- genetics
- Virus Replication -- drug effects
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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