A novel Rac-dependent checkpoint in B cell development controls entry into the splenic white pulp and cell survival. [electronic resource]
Producer: 20100506Description: 837-53 p. digitalISSN:- 1540-9538
- Animals
- Antibodies -- immunology
- Antigens, CD -- metabolism
- B-Lymphocyte Subsets -- cytology
- B-Lymphocytes -- cytology
- Bone Marrow Cells -- cytology
- Cell Adhesion -- genetics
- Cell Differentiation -- drug effects
- Cell Movement -- drug effects
- Cell Proliferation
- Cell Survival -- genetics
- Chemokines -- pharmacology
- Immunoglobulin D -- metabolism
- Integrins -- antagonists & inhibitors
- Intracellular Signaling Peptides and Proteins -- genetics
- Lymphocyte Function-Associated Antigen-1 -- metabolism
- Mice
- Mice, Inbred C3H
- Mice, Inbred C57BL
- Mice, Knockout
- Mice, Mutant Strains
- Mice, Transgenic
- Neuropeptides -- genetics
- Pertussis Toxin -- pharmacology
- Protein-Tyrosine Kinases -- genetics
- Proto-Oncogene Proteins c-vav -- genetics
- Receptors, CXCR4 -- antagonists & inhibitors
- Receptors, Chemokine -- antagonists & inhibitors
- Signal Transduction -- drug effects
- Spleen -- cytology
- Syk Kinase
- bcl-X Protein -- genetics
- rac GTP-Binding Proteins -- genetics
- rac1 GTP-Binding Protein
- rap1 GTP-Binding Proteins -- metabolism
- RAC2 GTP-Binding Protein
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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