Nicotine restores endothelial dysfunction caused by excess sFlt1 and sEng in an in vitro model of preeclamptic vascular endothelium: a possible therapeutic role of nicotinic acetylcholine receptor (nAChR) agonists for preeclampsia. [electronic resource]
Producer: 20100601Description: 464.e1-6 p. digitalISSN:- 1097-6868
- Antigens, CD -- physiology
- Endoglin
- Endothelium, Vascular -- drug effects
- Enzyme-Linked Immunosorbent Assay
- Female
- Humans
- Nicotine -- pharmacology
- Nicotinic Agonists -- pharmacology
- Pre-Eclampsia -- drug therapy
- Pregnancy
- Receptors, Cell Surface -- physiology
- Receptors, Cholinergic -- drug effects
- Receptors, Nicotinic -- drug effects
- Vascular Endothelial Growth Factor Receptor-1 -- physiology
- Wound Healing -- physiology
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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