AMPKalpha2 deletion causes aberrant expression and activation of NAD(P)H oxidase and consequent endothelial dysfunction in vivo: role of 26S proteasomes. [electronic resource]
Producer: 20100426Description: 1117-28 p. digitalISSN:- 1524-4571
- AMP-Activated Protein Kinases -- antagonists & inhibitors
- Acetophenones -- pharmacology
- Acetylcholine -- pharmacology
- Aminoimidazole Carboxamide -- analogs & derivatives
- Animals
- Antioxidants -- pharmacology
- Cells, Cultured
- Cyclic N-Oxides -- pharmacology
- Dose-Response Relationship, Drug
- Endothelial Cells -- drug effects
- Endothelium, Vascular -- drug effects
- Enzyme Activation
- Enzyme Inhibitors -- pharmacology
- Gene Deletion
- Gene Expression Regulation, Enzymologic
- Humans
- I-kappa B Proteins -- metabolism
- Inflammation Mediators -- blood
- Interferon-gamma -- blood
- Interleukin-2 -- blood
- Isoenzymes
- Leupeptins -- pharmacology
- Male
- Mice
- Mice, Inbred C57BL
- Mice, Knockout
- NADPH Oxidases -- antagonists & inhibitors
- NF-KappaB Inhibitor alpha
- NF-kappa B -- metabolism
- Nitroprusside -- pharmacology
- Oxidative Stress -- drug effects
- Proteasome Endopeptidase Complex -- metabolism
- Proteasome Inhibitors
- RNA Interference
- Receptors, LDL -- deficiency
- Ribonucleotides -- pharmacology
- Spin Labels
- Superoxide Dismutase -- metabolism
- Superoxides -- metabolism
- Transfection
- Vasodilation -- drug effects
- Vasodilator Agents -- pharmacology
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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