Nitric oxide induces pathological synapse loss by a protein kinase G-, Rho kinase-dependent mechanism preceded by myosin light chain phosphorylation. [electronic resource]
Producer: 20100205Description: 973-84 p. digitalISSN:- 1529-2401
- Analysis of Variance
- Animals
- Animals, Newborn
- Brain Stem -- cytology
- Cyclic GMP-Dependent Protein Kinases -- antagonists & inhibitors
- DNA-Binding Proteins -- genetics
- Enzyme Inhibitors -- pharmacology
- Green Fluorescent Proteins -- genetics
- Humans
- Hypoglossal Nerve Diseases -- pathology
- In Vitro Techniques
- Male
- Microscopy, Immunoelectron -- methods
- Motor Neurons -- drug effects
- Myosin Light Chains -- metabolism
- Nitric Oxide -- pharmacology
- Nitric Oxide Synthase Type I -- genetics
- Nuclear Proteins -- genetics
- Patch-Clamp Techniques
- Phosphorylation -- drug effects
- Presynaptic Terminals -- drug effects
- Rats
- Rats, Wistar
- Synapses -- drug effects
- Synaptic Potentials -- drug effects
- Synaptophysin -- metabolism
- Transfection
- Vesicular Glutamate Transport Protein 2 -- metabolism
- Vesicular Inhibitory Amino Acid Transport Proteins -- metabolism
- rho-Associated Kinases -- antagonists & inhibitors
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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