Bmi1 functions as an oncogene independent of Ink4A/Arf repression in hepatic carcinogenesis. [electronic resource]
Producer: 20100219Description: 1937-45 p. digitalISSN:- 1557-3125
- Animals
- Carcinoma, Hepatocellular -- genetics
- Cell Cycle
- Cell Line, Tumor
- Cell Proliferation
- Cyclin-Dependent Kinase Inhibitor p16 -- genetics
- Down-Regulation -- genetics
- Enzyme Activation
- Gene Expression Regulation, Neoplastic
- Gene Knockdown Techniques
- Hepatocytes -- metabolism
- Humans
- Liver Neoplasms -- genetics
- Mice
- Nuclear Proteins -- genetics
- Polycomb Repressive Complex 1
- Proto-Oncogene Mas
- Proto-Oncogene Proteins -- genetics
- RNA, Small Interfering -- metabolism
- Repressor Proteins -- genetics
- Tumor Suppressor Protein p14ARF -- genetics
- Up-Regulation -- genetics
- ras Proteins -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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