Sustained deficiency of mitochondrial complex I activity during long periods of survival after seizures induced in immature rats by homocysteic acid. [electronic resource]
Producer: 20100608Description: 394-403 p. digitalISSN:- 1872-9754
- Aldehydes -- metabolism
- Animals
- Animals, Newborn
- Cerebral Cortex -- metabolism
- Convulsants -- toxicity
- Disease Models, Animal
- Down-Regulation -- drug effects
- Electron Transport Complex I -- drug effects
- Energy Metabolism -- drug effects
- Epilepsy -- metabolism
- Excitatory Amino Acid Agonists -- pharmacology
- Free Radical Scavengers -- pharmacology
- Homocysteine -- analogs & derivatives
- Male
- Metabolic Networks and Pathways -- physiology
- Mitochondria -- drug effects
- Mitochondrial Diseases -- chemically induced
- Oxidative Stress -- drug effects
- Rats
- Rats, Wistar
- Seizures -- chemically induced
- Survival Rate
- Time Factors
- Tyrosine -- analogs & derivatives
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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