RAS mutations affect tumor necrosis factor-induced apoptosis in colon carcinoma cells via ERK-modulatory negative and positive feedback circuits along with non-ERK pathway effects. [electronic resource]
Producer: 20091124Description: 8191-9 p. digitalISSN:- 1538-7445
- Apoptosis -- drug effects
- Autocrine Communication
- Blotting, Western
- Chemokines
- Colonic Neoplasms -- genetics
- Computational Biology
- Dual-Specificity Phosphatases -- genetics
- Enzyme-Linked Immunosorbent Assay
- Extracellular Signal-Regulated MAP Kinases -- metabolism
- Feedback, Physiological
- Flow Cytometry
- Fluorescent Antibody Technique
- Genes, ras -- genetics
- Humans
- Mutation -- genetics
- Proto-Oncogene Proteins -- genetics
- Proto-Oncogene Proteins p21(ras)
- RNA, Messenger -- genetics
- Reverse Transcriptase Polymerase Chain Reaction
- Transforming Growth Factor beta -- genetics
- Tumor Cells, Cultured
- Tumor Necrosis Factor-alpha -- pharmacology
- ras Proteins -- genetics
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Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
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