Atorvastatin restores the impaired vascular endothelium-dependent relaxations mediated by nitric oxide and endothelium-derived hyperpolarizing factors but not hypotension in sepsis. [electronic resource]
Producer: 20100426Description: 526-34 p. digitalISSN:- 1533-4023
- Acetylcholine -- pharmacology
- Animals
- Aorta -- drug effects
- Atorvastatin
- Biological Factors -- antagonists & inhibitors
- Blood Cell Count
- Blood Pressure -- drug effects
- Calcium Channel Blockers -- pharmacology
- Cytochrome P-450 Enzyme Inhibitors
- Disease Models, Animal
- Endothelium, Vascular -- drug effects
- Gene Expression -- drug effects
- Heptanoic Acids -- pharmacology
- Hydroxymethylglutaryl-CoA Reductase Inhibitors -- pharmacology
- Hypotension -- etiology
- Indomethacin -- pharmacology
- Male
- NG-Nitroarginine Methyl Ester -- pharmacology
- Nitric Oxide -- antagonists & inhibitors
- Nitric Oxide Synthase Type II -- antagonists & inhibitors
- Nitric Oxide Synthase Type III -- antagonists & inhibitors
- Nitrites -- blood
- Nitroprusside -- pharmacology
- Phenylephrine -- pharmacology
- Pulmonary Artery -- drug effects
- Pyrroles -- pharmacology
- Rats
- Rats, Wistar
- Sepsis -- blood
- Vasodilation -- drug effects
No physical items for this record
Publication Type: Journal Article
There are no comments on this title.
Log in to your account to post a comment.