CaMKII phosphorylates collapsin response mediator protein 2 and modulates axonal damage during glutamate excitotoxicity. [electronic resource]
Producer: 20091106Description: 870-81 p. digitalISSN:- 1471-4159
- Animals
- Axons -- drug effects
- Brain -- metabolism
- Calcium-Calmodulin-Dependent Protein Kinase Type 2 -- metabolism
- Cell Death -- drug effects
- Cells, Cultured
- Cerebral Cortex -- cytology
- Disease Models, Animal
- Dizocilpine Maleate -- pharmacology
- Embryo, Mammalian
- Enzyme Activation -- drug effects
- Enzyme Inhibitors -- pharmacology
- Excitatory Amino Acid Antagonists -- pharmacology
- Glutamic Acid -- pharmacology
- Green Fluorescent Proteins -- genetics
- Infarction, Middle Cerebral Artery -- pathology
- Intercellular Signaling Peptides and Proteins -- genetics
- Mice
- Mice, Inbred C57BL
- Mutation -- genetics
- Nerve Tissue Proteins -- genetics
- Neurons -- cytology
- Phosphorylation -- drug effects
- Semaphorin-3A -- pharmacology
- Signal Transduction -- drug effects
- Transfection -- methods
- Tubulin -- metabolism
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Publication Type: Journal Article; Research Support, Non-U.S. Gov't
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