Beta-amyloid overload does not directly correlate with SAPK/JNK activation and tau protein phosphorylation in the cerebellar cortex of Ts65Dn mice. [electronic resource]
Producer: 20100108Description: 198-206 p. digitalISSN:- 1872-6240
- Amyloid beta-Peptides -- genetics
- Animals
- Axons -- metabolism
- Cerebellum -- metabolism
- Disease Models, Animal
- Down Syndrome -- genetics
- Female
- Genetic Predisposition to Disease -- genetics
- Interneurons -- metabolism
- JNK Mitogen-Activated Protein Kinases -- metabolism
- Male
- Mice
- Mice, Transgenic
- Mitogen-Activated Protein Kinase 8 -- metabolism
- Nerve Degeneration -- genetics
- Neuroglia -- metabolism
- Phosphorylation
- Purkinje Cells -- metabolism
- Signal Transduction -- physiology
- Tauopathies -- genetics
- Trisomy -- genetics
- tau Proteins -- metabolism
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Publication Type: Journal Article
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