APA

Gourronc F. A., Robertson m. M., Herrig A. K., Lansdorp P. M., Goldman F. D. & Klingelhutz A. J. (20100629). Proliferative defects in dyskeratosis congenita skin keratinocytes are corrected by expression of the telomerase reverse transcriptase, TERT, or by activation of endogenous telomerase through expression of papillomavirus E6/E7 or the telomerase RNA component, TERC. : Experimental dermatology.

Chicago

Gourronc Francoise A, Robertson mckaylee M, Herrig Annie K, Lansdorp Peter M, Goldman Frederick D and Klingelhutz Aloysius J. 20100629. Proliferative defects in dyskeratosis congenita skin keratinocytes are corrected by expression of the telomerase reverse transcriptase, TERT, or by activation of endogenous telomerase through expression of papillomavirus E6/E7 or the telomerase RNA component, TERC. : Experimental dermatology.

Harvard

Gourronc F. A., Robertson m. M., Herrig A. K., Lansdorp P. M., Goldman F. D. and Klingelhutz A. J. (20100629). Proliferative defects in dyskeratosis congenita skin keratinocytes are corrected by expression of the telomerase reverse transcriptase, TERT, or by activation of endogenous telomerase through expression of papillomavirus E6/E7 or the telomerase RNA component, TERC. : Experimental dermatology.

MLA

Gourronc Francoise A, Robertson mckaylee M, Herrig Annie K, Lansdorp Peter M, Goldman Frederick D and Klingelhutz Aloysius J. Proliferative defects in dyskeratosis congenita skin keratinocytes are corrected by expression of the telomerase reverse transcriptase, TERT, or by activation of endogenous telomerase through expression of papillomavirus E6/E7 or the telomerase RNA component, TERC. : Experimental dermatology. 20100629.