Prolyl hydroxylase inhibition attenuates post-ischemic cardiac injury via induction of endoplasmic reticulum stress genes. [electronic resource]
Producer: 20091030Description: 110-8 p. digitalISSN:- 1879-3649
- Activating Transcription Factor 4 -- genetics
- Amino Acids, Dicarboxylic -- pharmacology
- Analysis of Variance
- Animals
- Apoptosis -- drug effects
- Biomarkers -- metabolism
- Caspase 3 -- metabolism
- Cell Hypoxia -- drug effects
- Cell Line
- DNA-Binding Proteins -- genetics
- Endoplasmic Reticulum -- drug effects
- Endoplasmic Reticulum Chaperone BiP
- Endothelial Cells -- drug effects
- Enzyme Activation
- Enzyme Inhibitors -- pharmacology
- Gene Expression Regulation -- drug effects
- Gene Silencing
- Heat-Shock Proteins -- genetics
- Heme Oxygenase-1 -- genetics
- Male
- Mice
- Microscopy, Fluorescence
- Microvessels
- Myocardial Reperfusion Injury -- drug therapy
- Myocardium -- metabolism
- Myocytes, Cardiac -- drug effects
- NF-E2-Related Factor 2 -- immunology
- Phosphorylation
- Procollagen-Proline Dioxygenase -- antagonists & inhibitors
- Protein Transport
- RNA, Messenger -- metabolism
- RNA, Small Interfering
- Regulatory Factor X Transcription Factors
- Reverse Transcriptase Polymerase Chain Reaction
- Stress, Physiological -- drug effects
- Transcription Factor CHOP -- genetics
- Transcription Factors -- genetics
- eIF-2 Kinase -- metabolism
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Publication Type: Journal Article; Research Support, N.I.H., Extramural
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